Abstract
Tracer studies with the use of 2-14C-acetate revealed that the infected tissue of diseased sweet potato roots with black rot was incapable of synthesizing ipomeamarone from acetate, but was capable of synthesizing it from some intermediate(s) which was produced from acetate by the non-infected tissue in a short period. It is very likely that migration of the intermediate(s) from the non-infected tissue to the infected region occurs during infection process. Significance of the intermediate(s) in ipomeamarone accumulation was discussed.

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