Chronic pre-treatment with nicotine enhances nicotine-evoked striatal dopamine release and α6 and β3 nicotinic acetylcholine receptor subunit mRNA in the substantia nigra pars compacta of the rat
- 1 January 2006
- journal article
- Published by Elsevier in Neuropharmacology
- Vol. 50 (1) , 36-46
- https://doi.org/10.1016/j.neuropharm.2005.07.013
Abstract
No abstract availableKeywords
This publication has 37 references indexed in Scilit:
- The β3 Nicotinic Receptor Subunit: A Component of α-Conotoxin MII-Binding Nicotinic Acetylcholine Receptors that Modulate Dopamine Release and Related BehaviorsJournal of Neuroscience, 2003
- Subunit Composition of Functional Nicotinic Receptors in Dopaminergic Neurons Investigated with Knock-Out MiceJournal of Neuroscience, 2003
- Expression and functional characterisation of a human chimeric nicotinic receptor with α6β4 propertiesEuropean Journal of Pharmacology, 2003
- effect of unilateral 6-hydroxydopamine lesions of the nigrostriatal pathway on GABAA receptor subunit gene expression in the rodent basal ganglia and thalamusNeuroscience, 1999
- Regional variation in the effects of nicotine on catecholamine overflow in rat brainEuropean Journal of Pharmacology, 1997
- Pharmacology of nicotine and its therapeutic use in smoking cessation and neurodegenerative disordersPharmacology & Therapeutics, 1996
- Comparative Alterations of Nicotinic and Muscarinic Binding Sites in Alzheimer's and Parkinson's DiseasesJournal of Neurochemistry, 1992
- Differential Effect of Stress on In Vivo Dopamine Release in Striatum, Nucleus Accumbens, and Medial Frontal CortexJournal of Neurochemistry, 1989
- Drugs abused by humans preferentially increase synaptic dopamine concentrations in the mesolimbic system of freely moving rats.Proceedings of the National Academy of Sciences, 1988
- Evidence that Tobacco Smoking Increases the Density of (−)‐[3H]Nicotine Binding Sites in Human BrainJournal of Neurochemistry, 1988