THE MOLECULAR PATHOPHYSIOLOGY OF MYELOID LEUKAEMIAS: Ras REVISITED
Open Access
- 1 February 1998
- journal article
- review article
- Published by Wiley in British Journal of Haematology
- Vol. 100 (2) , 256-264
- https://doi.org/10.1046/j.1365-2141.1998.00554.x
Abstract
No abstract availableKeywords
This publication has 62 references indexed in Scilit:
- Role of CED-4 in the activation of CED-3Nature, 1997
- Loss of NF1 results in activation of the Ras signaling pathway and leads to aberrant growth in haematopoietic cellsNature Genetics, 1996
- Structural and Signaling Requirements for BCR-ABL-Mediated Transformation and Inhibition of ApoptosisMolecular and Cellular Biology, 1995
- Protein kinase B (c-Akt) in phosphatidylinositol-3-OH kinase signal transductionNature, 1995
- The pathway to signal achievementNature, 1993
- Analysis of RAS gene mutations in acute myeloid leukemia by polymerase chain reaction and oligonucleotide probes.Proceedings of the National Academy of Sciences, 1988
- ras GENESAnnual Review of Biochemistry, 1987
- Microinjection of the ras oncogene protein into PC12 cells induces morphological differentiationCell, 1985
- Amino-acid substitutions at codon 13 of the N-ras oncogene in human acute myeloid leukaemiaNature, 1985
- Transforming genes of human hematopoietic tumors: frequent detection of ras-related oncogenes whose activation appears to be independent of tumor phenotype.Proceedings of the National Academy of Sciences, 1983