Shutoff of Histone Synthesis by Mengovirus Infection of Ehrlich Ascites Tumor Cells

Abstract
The histone synthesizing capacity of mengovirus-infected [mouse] Ehrlich ascites tumor cells and their corresponding postnuclear supernatants was investigated as a function of post-infection time. Histone synthesis was compared with the synthesis of other basic host proteins under identical conditions. For mengovirus infection of Ehrlich ascites tumor cells, the less complex fraction comprising basic protein, separated from the acidic proteins by CM-cellulose chromatography, represents total host protein. Histones and the remaining basic host proteins are easily identifiable indicators of the host protein synthesizing potential. The cessation of histone synthesis proceeds faster than that of the synthesis of other basic host protein.

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