A polymorphism within intron 11 of the tau gene is not increased in frequency in patients with sporadic Alzheimer's disease, nor does it influence the extent of tau pathology in the brain
- 17 May 2002
- journal article
- research article
- Published by Elsevier in Neuroscience Letters
- Vol. 324 (2) , 113-116
- https://doi.org/10.1016/s0304-3940(02)00190-8
Abstract
No abstract availableKeywords
This publication has 27 references indexed in Scilit:
- No association between TAU haplotype and Alzheimer's disease in population or clinic based series or in familial diseaseNeuroscience Letters, 2000
- A polymorphism in the tau gene associated with risk for Alzheimer's diseaseNeuroscience Letters, 2000
- Identification of a novel polymorphism in the promoter region of the tau gene highly associated to progressive supranuclear palsy in humansNeuroscience Letters, 1999
- No genetic association between polymorphisms in the Tau gene and Alzheimer's disease in clinic or population based samplesNeuroscience Letters, 1999
- Association of an Extended Haplotype in the Tau Gene with Progressive Supranuclear PalsyHuman Molecular Genetics, 1999
- Direct genetic evidence for involvement of tau in progressive supranuclear palsyNeurology, 1998
- Progressive supranuclear gaze palsy is in linkage disequilibrium with theτ and not the α-synuclein geneNeurology, 1998
- Genetic evidence for the involvement of τ in progressive supranuclear palsyAnnals of Neurology, 1997
- Aβ‐peptide length aid apolipoprotein E genotype in Alzheimer's diseaseAnnals of Neurology, 1996
- Occurrence of neuropil threads in the senile human brain and in Alzheimer's disease: A third location of paired helical filaments outside of neurofibrillary tangles and neuritic plaquesNeuroscience Letters, 1986