Suppression of Calcium Release from Inositol 1,4,5-Trisphosphate-sensitive Stores Mediates the Anti-apoptotic Function of Nuclear Factor-κB
Open Access
- 1 June 2005
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 280 (23) , 22287-22296
- https://doi.org/10.1074/jbc.m410923200
Abstract
No abstract availableKeywords
This publication has 69 references indexed in Scilit:
- Bcl-2 increases emptying of endoplasmic reticulum Ca2+ stores during photodynamic therapy-induced apoptosisCell Calcium, 2001
- Homocysteine Induces Programmed Cell Death in Human Vascular Endothelial Cells through Activation of the Unfolded Protein ResponseJournal of Biological Chemistry, 2001
- Presenilin mutations and calcium signaling defects in the nervous and immune systemsBioEssays, 2001
- Pro-apoptotic action of PAR-4 involves inhibition of NF-?B activity and suppression of BCL-2 expressionJournal of Neuroscience Research, 2000
- Selective down-regulation of IP3receptor subtypes by caspases and calpain during TNF α -induced apoptosis of human T-lymphoma cellsCell Calcium, 2000
- Control of apoptosis by Rel/NF-κB transcription factorsOncogene, 1999
- TUMOR NECROSIS FACTOR RECEPTOR AND Fas SIGNALING MECHANISMSAnnual Review of Immunology, 1999
- Calcium signaling and cytotoxicity.Environmental Health Perspectives, 1999
- Signaling Mechanisms in Growth Factor‐Stimulated Cell MotilityThe International Journal of Cell Cloning, 1997
- Tumor necrosis factors protect neurons against metabolic-excitotoxic insults and promote maintenance of calcium homeostasisNeuron, 1994