The fusion gene Cbfb-MYH11 blocks myeloid differentiation and predisposes mice to acute myelomonocytic leukaemia

This publication has 18 references indexed in Scilit:

• Oncogenic Transcription Factors in the Human Acute Leukemias
  Science, 1997
• Embryonic lethality and impairment of haematopoiesis in mice heterozygous for an AML1-ETO fusion gene
  Nature Genetics, 1997
• Failure of Embryonic Hematopoiesis andLethal Hemorrhages in Mouse Embryos Heterozygousfor a Knocked-In Leukemia Gene CBFB–MYH11
  Cell, 1996
• The CBFβ Subunit Is Essential for CBFα2 (AML1) Function In Vivo
  Cell, 1996
• Multicolour spectral karyotyping of mouse chromosomes
  Nature Genetics, 1996
• Disruption of the Cbfa2 gene causes necrosis and hemorrhaging in the central nervous system and blocks definitive hematopoiesis.
  Proceedings of the National Academy of Sciences, 1996
• AML1, the Target of Multiple Chromosomal Translocations in Human Leukemia, Is Essential for Normal Fetal Liver Hematopoiesis
  Cell, 1996
• Fusion Between Transcription Factor CBFβ/PEBP2β and a Myosin Heavy Chain in Acute Myeloid Leukemia
  Science, 1993
• Cloning and characterization of subunits of the T-cell receptor and murine leukemia virus enhancer core-binding factor.
  Molecular and Cellular Biology, 1993
• Specific-locus test shows ethylnitrosourea to be the most potent mutagen in the mouse.
  Proceedings of the National Academy of Sciences, 1979