Nitric oxide blocks bile canalicular contraction by inhibiting inositol trisphosphate-dependent calcium mobilization
Open Access
- 31 March 1995
- journal article
- Published by Elsevier in Gastroenterology
- Vol. 108 (3) , 841-849
- https://doi.org/10.1016/0016-5085(95)90459-x
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- The nitric oxide and cGMP signal transduction system: regulation and mechanism of actionBiochimica et Biophysica Acta (BBA) - Molecular Cell Research, 1993
- Inositol 1,4,5-Trisphosphate-Activated Calcium ChannelsAnnual Review of Physiology, 1992
- INOSITOL PHOSPHATE BIOCHEMISTRYAnnual Review of Biochemistry, 1992
- Extracellular ATP, intracellular calcium and canalicular contraction in rat hepatocyte doubletsHepatology, 1991
- Cyclic Nucleotide Content of the Rat Anococcygeus During Relaxations Induced by Drugs or by Non-adrenergic, Non-cholinergic Field StimulationJournal of Pharmacy and Pharmacology, 1991
- Studies of the Effect of Glyceryl Trinitrate and Cyclic GMP on Calcium Turnover in Bovine Mesenteric ArteryBasic & Clinical Pharmacology & Toxicology, 1990
- Inositol phosphates and cell signallingNature, 1989
- PDGF-dependent tyrosine phosphorylation stimulates production of novel polyphosphoinositides in intact cellsCell, 1989
- Contractile activity of bile canaliculi: Contraction or collapse?Hepatology, 1987
- Inhibitory action of cyclic GMP on secretion, polyphosphoinositide hydrolysis and calcium mobilization in thrombin-stimulated human plateletsBiochemical and Biophysical Research Communications, 1986