K-ras codon 12 and 61 point mutations in bromodeoxyuridine- and N-nitrosomethylurea-induced rat renal mesenchymal tumors
- 3 December 1996
- journal article
- Published by Elsevier in Cancer Letters
- Vol. 109 (1-2) , 1-7
- https://doi.org/10.1016/s0304-3835(96)04350-9
Abstract
No abstract availableKeywords
This publication has 12 references indexed in Scilit:
- Effect of neonatal exposure to 5-bromo-2′-deoxyuridine on life span, estrus function and tumor development in rats — an argument in favor of the mutation theory of aging?Mutation Research/DNAging, 1992
- Analysis of sequence specificity of 5-bromodeoxyuridine-induced reversion in cells containing multiple copies of a mutantgpt geneSomatic Cell and Molecular Genetics, 1992
- Activating point mutation in Ki-ras codon 63 in a chemically induced rat renal tumorMolecular Carcinogenesis, 1992
- Effects of flanking base sequences on 5-bromodeoxyuridine mutagenesis in mammalian cellsSomatic Cell and Molecular Genetics, 1991
- Dietary N‐3 fatty acids do not affect induction of Ha‐ras mutations in mammary glands of NMU‐treated ratsMolecular Carcinogenesis, 1991
- Ki‐ras mutations in spontaneous and chemically induced renal tumors of the ratMolecular Carcinogenesis, 1991
- The role of Ha-ras oncogenes in growth factor independence in rat mammary carcinoma cellsMolecular Carcinogenesis, 1991
- Activation of ras Oncogenes Preceding the Onset of NeoplasiaScience, 1990
- Specific patterns of oncogene activation in transplacentally induced tumors.Proceedings of the National Academy of Sciences, 1990
- Direct mutagenesis of Ha-ras-1 oncogenes by N-nitroso-N-methylurea during initiation of mammary carcinogenesis in ratsNature, 1985