The Enzymatic Activity of CEM15/Apobec-3G Is Essential for the Regulation of the Infectivity of HIV-1 Virion but Not a Sole Determinant of Its Antiviral Activity

This publication has 31 references indexed in Scilit:

• DNA Deamination Mediates Innate Immunity to Retroviral Infection
  Cell, 2003
• The cytidine deaminase CEM15 induces hypermutation in newly synthesized HIV-1 DNA
  Nature, 2003
• Broad antiretroviral defence by human APOBEC3G through lethal editing of nascent reverse transcripts
  Nature, 2003
• RNA Editing Enzyme APOBEC1 and Some of Its Homologs Can Act as DNA Mutators
  Molecular Cell, 2002
• Isolation of a human gene that inhibits HIV-1 infection and is suppressed by the viral Vif protein
  Nature, 2002
• An Anthropoid-Specific Locus of Orphan C to U RNA-Editing Enzymes on Chromosome 22
  Genomics, 2002
• Both Amino- and Carboxyl-Terminal Domains of TRAF3 Negatively Regulate NF-κB Activation Induced by OX40 Signaling
  Biochemical and Biophysical Research Communications, 2000
• Escherichia coli cytidine deaminase provides a molecular model for ApoB RNA editing and a mechanism for RNA substrate recognition 1 1Edited by A. R. Fersht
  Journal of Molecular Biology, 1998
• Evolutionary origins of apoB mRNA editing: Catalysis by a cytidine deaminase that has acquired a novel RNA-binding motif at its active site
  Cell, 1995
• Molecular Cloning of an Apolipoprotein B Messenger RNA Editing Protein
  Science, 1993