Ro 40-5967, a novel calcium channel antagonist, protects against ventricular fibrillation

This publication has 33 references indexed in Scilit:

• Electrocardiographic subset analysis of diltiazem administration on long-term outcome after acute myocardial infarction
  The American Journal of Cardiology, 1991
• Effect of verapamil on mortality and major events after acute myocardial infarction (The Danish Verapamil Infarction Trial II — DAVIT II)
  The American Journal of Cardiology, 1990
• The structurally novel Ca2+ channel blocker Ro 40-5967, which binds to the [3H] desmethoxyverapamil receptor, is devoid of the negative inotropic effects of verapamil in normal and failing rat hearts
  Cardiovascular Drugs and Therapy, 1990
• Effects of Ro 40-5967, a Novel Calcium Antagonist, on Myocardial Function During Ischemia Induced by Lowering Coronary Perfusion Pressure in Dogs: Comparison with Verapamil
  Journal of Cardiovascular Pharmacology, 1989
• The effects of daily exercise on susceptibility to sudden cardiac death.
  Circulation, 1984
• Autonomic mechanisms in ventricular fibrillation induced by myocardial ischemia during exercise in dogs with healed myocardial infarction. An experimental preparation for sudden cardiac death.
  Circulation, 1984
• The relationships among ventricular arrhythmias, left ventricular dysfunction, and mortality in the 2 years after myocardial infarction.
  Circulation, 1984
• Baroreceptor reflex control of heart rate: a predictor of sudden cardiac death.
  Circulation, 1982
• Effects of verapamil on ventricular rhythm during acute coronary occlusion
  American Heart Journal, 1978
• Electrical Stability of Acutely Ischemic Myocardium
  Circulation, 1973