Lessons from models of SOD1-linked familial ALS
- 1 August 2004
- journal article
- review article
- Published by Elsevier in Trends in Molecular Medicine
- Vol. 10 (8) , 393-400
- https://doi.org/10.1016/j.molmed.2004.06.009
Abstract
No abstract availableKeywords
This publication has 75 references indexed in Scilit:
- Cell death in amyotrophic lateral sclerosis: interplay between neuronal and glial cellsThe FASEB Journal, 2004
- Copper-binding-site-null SOD1 causes ALS in transgenic mice: aggregates of non-native SOD1 delineate a common featureHuman Molecular Genetics, 2003
- Amyloid-like filaments and water-filled nanotubes formed by SOD1 mutant proteins linked to familial ALSNature Structural & Molecular Biology, 2003
- MitochondriaCell, 2003
- Analysis of the Cytosolic Proteome in a Cell Culture Model of Familial Amyotrophic Lateral Sclerosis Reveals Alterations to the Proteasome, Antioxidant Defenses, and Nitric Oxide Synthetic PathwaysJournal of Biological Chemistry, 2003
- Oxidative modulation of nuclear factor‐κB in human cells expressing mutant fALS‐typical superoxide dismutasesJournal of Neurochemistry, 2002
- Expression of glutamate receptor subtypes in the spinal cord of control andmnd mice, a model of motor neuron disorderJournal of Neuroscience Research, 2002
- Of mice, men and motor neuronsTrends in Molecular Medicine, 2002
- Superoxide dismutase and the death of motoneurons in ALSTrends in Neurosciences, 2001
- Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosisNature, 1993