Does activation of the innate immune system contribute to the development of rheumatoid arthritis?

This publication has 36 references indexed in Scilit:

• Reduction of CpG‐induced arthritis by suppressive oligodeoxynucleotides
  Arthritis & Rheumatism, 2002
• Chromatin–IgG complexes activate B cells by dual engagement of IgM and Toll-like receptors
  Nature, 2002
• CpG Stimulation of Primary Mouse B Cells Is Blocked by Inhibitory Oligodeoxyribonucleotides at a Site Proximal to NF-κB Activation
  Antisense and Nucleic Acid Drug Development, 2001
• Presence of bacterial DNA and bacterial peptidoglycans in joints of patients with rheumatoid arthritis and other arthritides
  Arthritis & Rheumatism, 2000
• The features of arthritis induced by CpG motifs in bacterial DNA
  Arthritis & Rheumatism, 2000
• Innate immunity: impact on the adaptive immune response
  Current Opinion in Immunology, 1997
• A Pathogenetic Role for TNFα in the Syndrome of Cachexia, Arthritis, and Autoimmunity Resulting from Tristetraprolin (TTP) Deficiency
  Immunity, 1996
• Disease severity in patients with systemic lupus erythematosus correlates with an increased ratio of interleukin‐10: Interferon‐γ–secreting cells in the peripheral blood
  Arthritis & Rheumatism, 1996
• CpG motifs in bacterial DNA trigger direct B-cell activation
  Nature, 1995
• Increased levels of soluble tumor necrosis factor receptors in the sera and synovial fluid of patients with rheumatic diseases
  Arthritis & Rheumatism, 1992