Protein trafficking abnormalities: a new mechanism in drug-induced long QT syndrome
Open Access
- 1 May 2005
- journal article
- editorial
- Published by Wiley in British Journal of Pharmacology
- Vol. 145 (1) , 3-4
- https://doi.org/10.1038/sj.bjp.0706143
Abstract
No abstract availableKeywords
This publication has 7 references indexed in Scilit:
- Pentamidine reduces hERG expression to prolong the QT intervalBritish Journal of Pharmacology, 2005
- Pentamidine-Induced Long QT Syndrome and Block of hERG TraffickingThe Journal of Pharmacology and Experimental Therapeutics, 2005
- Mechanisms of Arsenic-Induced Prolongation of Cardiac RepolarizationMolecular Pharmacology, 2004
- Biology of Cardiac ArrhythmiasCirculation Research, 2004
- Drug-Induced Prolongation of the QT IntervalNew England Journal of Medicine, 2004
- Unusual Effects of a QT-Prolonging Drug, Arsenic Trioxide, on Cardiac Potassium CurrentsCirculation, 2004
- Correction of Defective Protein Trafficking of a Mutant HERG Potassium Channel in Human Long QT SyndromeJournal of Biological Chemistry, 1999