Evasion of Early Cellular Response Mechanisms following Low Level Radiation-induced DNA Damage
Open Access
- 1 November 2004
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 279 (48) , 49624-49632
- https://doi.org/10.1074/jbc.m409600200
Abstract
No abstract availableKeywords
This publication has 40 references indexed in Scilit:
- Distinct functional domains of Nbs1 modulate the timing and magnitude of ATM activation after low doses of ionizing radiationOncogene, 2004
- Low-Dose Hyper-radiosensitivity: A Consequence of Ineffective Cell Cycle Arrest of Radiation-Damaged G2-Phase CellsRadiation Research, 2004
- Protective packaging for DNANature, 2003
- An Association between the Radiation-Induced Arrest of G2-Phase Cells and Low-Dose Hyper-Radiosensitivity: A Plausible Underlying Mechanism?Radiation Research, 2003
- ATM and related protein kinases: safeguarding genome integrityNature Reviews Cancer, 2003
- DNA damage activates ATM through intermolecular autophosphorylation and dimer dissociationNature, 2003
- Genomic Instability in Mice Lacking Histone H2AXScience, 2002
- ATM Phosphorylates Histone H2AX in Response to DNA Double-strand BreaksJournal of Biological Chemistry, 2001
- Correlation of Tumor Sensitivity to Low-Dose-Rate Irradiation with G 2 /M-Phase Block and Other Radiobiological ParametersRadiation Research, 1993
- Response of glioblastoma cell lines to low dose rate irradiationInternational Journal of Radiation Oncology*Biology*Physics, 1991