Who are the proteolytic culprits in vascular disease?
Open Access
- 1 November 1999
- journal article
- editorial
- Published by American Society for Clinical Investigation in Journal of Clinical Investigation
- Vol. 104 (9) , 1167-1168
- https://doi.org/10.1172/jci8642
Abstract
No abstract availableThis publication has 24 references indexed in Scilit:
- Cystatin C deficiency in human atherosclerosis and aortic aneurysmsJournal of Clinical Investigation, 1999
- Gene transfer of tissue inhibitor of metalloproteinase-2 inhibits metalloproteinase activity and neointima formation in human saphenous veinsGene Therapy, 1998
- Local overexpression of TIMP-1 prevents aortic aneurysm degeneration and rupture in a rat model.Journal of Clinical Investigation, 1998
- Expression of the elastolytic cathepsins S and K in human atheroma and regulation of their production in smooth muscle cells.Journal of Clinical Investigation, 1998
- Adenovirus-Mediated Gene Transfer of the Human TIMP-1 Gene Inhibits Smooth Muscle Cell Migration and Neointimal Formation in Human Saphenous VeinHuman Gene Therapy, 1998
- Urokinase-generated plasmin activates matrix metalloproteinases during aneurysm formationNature Genetics, 1997
- Macrophages and atherosclerotic plaque stabilityCurrent Opinion in Lipidology, 1996
- Pycnodysostosis, a Lysosomal Disease Caused by Cathepsin K DeficiencyScience, 1996
- Inhibition of Matrix Metalloproteinase Activity Inhibits Smooth Muscle Cell Migration but Not Neointimal Thickening After Arterial InjuryCirculation Research, 1996
- Procathepsin L Degrades Extracellular Matrix Proteins in the Presence of Glycosaminoglycans in VitroBiochemical and Biophysical Research Communications, 1995