Effects of NMDA antagonists on picrotoxin-, low Mg2+- and low Ca2+-induced epileptogenesis and on evoked changes in extracellular Na+ and Ca2+ concentrations in rat hippocampal slices
- 31 December 1989
- journal article
- Published by Elsevier in Epilepsy Research
- Vol. 4 (3) , 187-200
- https://doi.org/10.1016/0920-1211(89)90003-x
Abstract
No abstract availableKeywords
This publication has 52 references indexed in Scilit:
- Ketamine blocks an NMDA receptor-mediated component of synaptic transmission in rat hippocampus in a voltage-dependent mannerNeuroscience Letters, 1988
- Selective inhibition of synaptic versus non-synaptic epileptogenesis by NMDA antagonists in the in vitro hippocampusEpilepsy Research, 1988
- Seizure-like discharges induced by lowering [Mg2+]0 in the human epileptogenic neocortex maintained in vitroBrain Research, 1987
- Magnesium-free medium activates seizure-like events in the rat hippocampal sliceBrain Research, 1986
- Magnesium ions block an N-methyl-d-aspartate receptor-mediated component of synaptic transmission in rat hippocampusNeuroscience Letters, 1985
- Anticonvulsant Action of Excitatory Amino Acid AntagonistsScience, 1982
- Stimulus-induced changes in extracellular Na+ and Cl− concentration in relation to changes in the size of the extracellular spaceExperimental Brain Research, 1982
- Selective depression of excitatory amino acid induced depolarizations by magnesium ions in isolated spinal cord preparations.The Journal of Physiology, 1980
- The role of calcium ions in tetanic and post‐tetanic increase of miniature end‐plate potential frequency.The Journal of Physiology, 1978
- Genesis of epileptic interictal spikes. New knowledge of cortical feedback systems suggests a neurophysiological explanation of brief paroxysmsBrain Research, 1973