Abstract
The period length of the circadian rhythm of phototactic response in Euglena is increased by cycloheximide, an inhibitor of protein synthesis. Furthermore, the effects of the drug are on the biological clock mechanism itself rather than on some parameter controlled by the clock or on some "coupling" mechanism between the clock and the parameter, as confirmed by experiments involving phase-shifting of the oscillation by light. The possible involvement of protein synthesis in the operation of the clock is discussed.

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