5-HT2C receptors inhibit and 5-HT1A receptors activate the generation of spike–wave discharges in a genetic rat model of absence epilepsy

This publication has 41 references indexed in Scilit:

• Regional distribution and cellular localization of 5‐HT_2C receptor mRNA in monkey brain: Comparison with [³H]mesulergine binding sites and choline acetyltransferase mRNA
  Synapse, 2001
• Effect of 5,7-dihydroxytryptamine on audiogenic seizures in genetically epilepsy-prone rats
  Life Sciences, 1996
• Targeting Epilepsy Genes
  Neuron, 1996
• Role of the hypothalamic paraventricular nucleus in 5-HT1A, 5-HT2A and 5-HT2C receptor-mediated oxytocin, prolactin and ACTH/corticosterone responses
  Behavioural Brain Research, 1995
• Eating disorder and epilepsy in mice lacking 5-HT2C serotonin receptors
  Nature, 1995
• Role of 5‐Hydroxytryptamine Receptor Subtypes in the 1‐[3‐(Trifluoromethyl)Phenyl] Piperazine‐Induced Increase in Threshold for Maximal Electroconvulsions in Mice
  Epilepsia, 1994
• Anticonvulsant Effect of Fluoxetine on Focally Evoked Limbic Motor Seizures in Rats
  Epilepsia, 1993
• Genetic models of absence epilepsy, with emphasis on the WAG/Rij strain of rats
  Epilepsy Research, 1992
• Effect of drugs modifying central serotonergic function on the response of extensor and nonextensor rats to maximal electroshock
  Life Sciences, 1978
• CHANGES IN SEIZURE SUSCEPTIBILITY AFTER INTRACEREBRAL TREATMENT WITH 5,7‐DIHYDROXYTRYPTAMINE: ROLE OF SEROTONERGIC NEURONS*
  Annals of the New York Academy of Sciences, 1978