Abstract
The survival responses and DNA repair abilities of AT [(ataxia telangiectasia) fibroblast] cells exposed to 4-nitroquinoline-1-oxide (4NQO), a chemical carcinogen whose DNA-damaging properties partially mimic those of ionizing radiation, were determined. Certain AT cell strains show hypersensitivity to inactivation by 4NQO and defective repair of 4NQO-induced adducts in DNA.

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