Chronic Pancreatitis Is Essential for Induction of Pancreatic Ductal Adenocarcinoma by K-Ras Oncogenes in Adult Mice
Top Cited Papers
Open Access
- 13 March 2007
- journal article
- research article
- Published by Elsevier
- Vol. 11 (3) , 291-302
- https://doi.org/10.1016/j.ccr.2007.01.012
Abstract
No abstract availableKeywords
This publication has 41 references indexed in Scilit:
- Smad4 is dispensable for normal pancreas development yet critical in progression and tumor biology of pancreas cancerGenes & Development, 2006
- Aggressive pancreatic ductal adenocarcinoma in mice caused by pancreas-specific blockade of transforming growth factor-β signaling in cooperation with active Kras expressionGenes & Development, 2006
- Hedgehog/Ras interactions regulate early stages of pancreatic cancerGenes & Development, 2006
- Nuclear factor-κB in cancer development and progressionNature, 2006
- Pathology of Genetically Engineered Mouse Models of Pancreatic Exocrine Cancer: Consensus Report and RecommendationsCancer Research, 2006
- Phosphorylation Meets Ubiquitination: The Control of NF-κB ActivityAnnual Review of Immunology, 2000
- NF-κB/Rel activation in cerulein pancreatitisGastroenterology, 1999
- Mice deficient for p53 are developmentally normal but susceptible to spontaneous tumoursNature, 1992
- Most human carcinomas of the exocrine pancreas contain mutant c-K-ras genesPublished by Elsevier ,1988
- Pancreatic neoplasia induced by ras expression in acinar cells of transgenic miceCell, 1987