Inhibition of complement activation by soluble recombinant CR1 under conditions resembling those in a cardiopulmonary circuit: reduced up-regulation of CD11b and complete abrogation of binding of PMNs to the biomaterial surface

This publication has 27 references indexed in Scilit:

• Complement activation during cardiopulmonary bypass: Effects of immobilized heparin
  The Annals of Thoracic Surgery, 1994
• Evidence for iC3 generation during cardiopulmonary bypass as the result of blood-gas interaction
  Clinical and Experimental Immunology, 1993
• Complement inhibition with soluble complement receptor type 1 in cardiopulmonary bypass
  The Annals of Thoracic Surgery, 1993
• Assessing the effect of cardiopulmonary bypass on the brain
  The Annals of Thoracic Surgery, 1991
• The response of human platelets to activated components of the complement system
  Immunology Today, 1991
• Formation of C5a during cardiopulmonary bypass: Inhibition by precoating with heparin
  The Annals of Thoracic Surgery, 1991
• Transient adhesion of neutrophils to endothelium.
  The Journal of Experimental Medicine, 1989
• Increased surface expression of CD11b/CD18 (Mac-1) is not required for stimulated neutrophil adherence to cultured endothelium.
  Journal of Clinical Investigation, 1988
• Identification of a C3bi-specific membrane complement receptor that is expressed on lymphocytes, monocytes, neutrophils, and erythrocytes
  The Journal of Experimental Medicine, 1982
• ISOLATION OF A FRAGMENT (C3a) OF THE THIRD COMPONENT OF HUMAN COMPLEMENT CONTAINING ANAPHYLATOXIN AND CHEMOTACTIC ACTIVITY AND DESCRIPTION OF AN ANAPHYLATOXIN INACTIVATOR OF HUMAN SERUM
  The Journal of Experimental Medicine, 1969