Homocysteine and Risk of Cardiovascular Disease Among Postmenopausal Women

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Abstract
Several mechanisms have been proposed linking hyperhomocystinemia to vascular damage, and it has been hypothesized that elevated levels of total plasma homocysteine represent an important modifiable risk factor for atherothrombotic disease.1,2 A number of retrospective and cross-sectional studies provide support for this hypothesis.3 However, because homocysteine levels may increase following acute myocardial infarction (MI)4 or stroke,5 any observed association could, at least in theory, be a result rather than a cause of acute vascular occlusion.

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