Hyperamylasemia in Critically Injured Patients
- 1 November 1980
- journal article
- research article
- Published by Wolters Kluwer Health
- Vol. 20 (11) , 951-955
- https://doi.org/10.1097/00005373-198011000-00008
Abstract
We found that in traumatic shock patients without pancreatic injury, hyperamylasemia occurs in high frequency [49 of 61 (80%)]. Isoenzyme studies of 19 of these patients revealed 18 of them (94%) to have the salivary type of hyperamylasemia. Further, based on the results of the present study, the salivary gland as the organ of origin and the permeability of its cell membranes are suggested as the mechanism for traumatic hyperamylasemia.This publication has 5 references indexed in Scilit:
- Susceptibility of the Pancreas to Ischemic Injury in ShockAnnals of Surgery, 1978
- The nature and origin of hyperamylasemia following open-heart surgery with extracorporeal circulationClinica Chimica Acta; International Journal of Clinical Chemistry, 1977
- Hyperamylasemia from the Binding of Serum Amylase by an 11S IgA GlobulinNew England Journal of Medicine, 1968
- Secretion and Synthesis of Amylase in the Rat Parotid Gland after IsoprenalineNature, 1966
- Amylase distribution in extrapancreatic, extrasalivary tissuesArchives of Biochemistry and Biophysics, 1958