A Protein C Deficiency Exacerbates Inflammatory and Hypotensive Responses in Mice During Polymicrobial Sepsis in a Cecal Ligation and Puncture Model
- 31 October 2004
- journal article
- Published by Elsevier in The American Journal of Pathology
- Vol. 165 (4) , 1433-1446
- https://doi.org/10.1016/s0002-9440(10)63401-3
Abstract
No abstract availableKeywords
Funding Information
- National Institutes of Health (HL19982, HL73750)
This publication has 77 references indexed in Scilit:
- Activated protein C signals through the thrombin receptor PAR1 in endothelial cellsInnate Immunity, 2003
- Activated protein C signals through the thrombin receptor PAR1 in endothelial cellsInnate Immunity, 2003
- Molecular crosstalk between p70S6k and MAPK cell signaling pathwaysBiochemical and Biophysical Research Communications, 2002
- Aging and ThrombosisSeminars in Thrombosis and Hemostasis, 2002
- Dysfunction of Endothelial Protein C Activation in Severe Meningococcal SepsisNew England Journal of Medicine, 2001
- Tissue Factor Pathway Inhibitor Does Not Influence Inflammatory Pathways during Human EndotoxemiaThe Journal of Infectious Diseases, 2001
- Disseminated Intravascular CoagulationNew England Journal of Medicine, 1999
- TAFI, or Plasma Procarboxypeptidase B, Couples the Coagulation and Fibrinolytic Cascades through the Thrombin-Thrombomodulin ComplexPublished by Elsevier ,1996
- Thrombin triggers the de novo expression of an inducible NO synthase in porcine aortic valve endothelial cellsEuropean Journal of Pharmacology: Molecular Pharmacology, 1995
- Detection of Circulating Tumor Necrosis Factor after Endotoxin AdministrationNew England Journal of Medicine, 1988