Infection with a periodontal pathogen increases mononuclear cell adhesion to human aortic endothelial cells
- 18 April 2006
- journal article
- Published by Elsevier
- Vol. 190 (2) , 271-281
- https://doi.org/10.1016/j.atherosclerosis.2006.03.018
Abstract
No abstract availableKeywords
This publication has 40 references indexed in Scilit:
- Porphyromonas gingivalisFimbria-Dependent Activation of Inflammatory Genes in Human Aortic Endothelial CellsInfection and Immunity, 2005
- Inflammation, Atherosclerosis, and Coronary Artery DiseaseNew England Journal of Medicine, 2005
- Effects of Porphyromonas gingivalis antigens and proinflammatory cytokines on human coronary artery endothelial cellsOral Microbiology and Immunology, 2005
- Porphyromonas gingivalisinduces its uptake by human macrophages and promotes foam cell formation in vitroFEMS Microbiology Letters, 2004
- Inflammation in atherosclerosisNature, 2002
- Porphyromonas gingivalisLipopolysaccharide Is Both Agonist and Antagonist for p38 Mitogen-Activated Protein Kinase ActivationInfection and Immunity, 2002
- The Association Between Periodontal Diseases and Cardiovascular Diseases: A State‐of‐the‐Science ReviewAnnals of Periodontology, 2001
- Chlamydia pneumoniae and the Risk of First Ischemic StrokeStroke, 2000
- Periodontal Diseases: EpidemiologyAnnals of Periodontology, 1996
- The Neutrophil-Activating Protein (NAP-1) Is Also Chemotactic for T LymphocytesScience, 1989