Reduction in Mitochondrial Membrane Potential Is an Early Event in Fas-Independent CTL-Mediated Apoptosis
- 10 July 1999
- journal article
- Published by Elsevier in Cellular Immunology
- Vol. 195 (1) , 43-52
- https://doi.org/10.1006/cimm.1999.1513
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- Granule-mediated Killing: Pathways for Granzyme B–initiated ApoptosisThe Journal of Experimental Medicine, 1997
- Apoptosis by Death FactorCell, 1997
- Programmed Cell Death in Animal DevelopmentCell, 1997
- Mitochondrial control of apoptosisPublished by Elsevier ,1997
- Why are mitochondria involved in apoptosis? Permeability transition pores and apoptosis as selective mechanisms to eliminate superoxide‐producing mitochondria and cellFEBS Letters, 1996
- Mitochondria and programmed cell death: back to the futureFEBS Letters, 1996
- Cytotoxic T-cell-derived granzyme B activates the apoptotic protease ICE-LAP3Current Biology, 1996
- ICE Family Proteases: Mediators of All Apoptotic Cell Death?Immunity, 1996
- Alterations in mitochondrial structure and function are early events of dexamethasone-induced thymocyte apoptosis.The Journal of cell biology, 1995
- A carbocyanine dye, DiOC6(3), acts as a mitochondrial probe in human neutrophilsBiochemical and Biophysical Research Communications, 1982