Redefinition of the coding sequence of the MXI1 gene and identification of a polymorphic repeat in the 3? non-coding region that allows the detection of loss of heterozygosity of chromosome 10q25 in glioblastomas
- 1 June 1995
- journal article
- Published by Springer Nature in Human Genetics
- Vol. 95 (6) , 709-711
- https://doi.org/10.1007/bf00209493
Abstract
No abstract availableKeywords
This publication has 9 references indexed in Scilit:
- Increasing complexity of the karyotype in 50 human gliomasCancer Genetics and Cytogenetics, 1994
- Mapping of two genes encoding members of a distinct subfamily of MAX interacting proteins: MAD to human chromosome 2 and mouse chromosome 6, and MXI1 to human chromosome 10 and mouse chromosome 19.1994
- Mad: A heterodimeric partner for Max that antagonizes Myc transcriptional activityCell, 1993
- Mxi1, a protein that specifically interacts with Max to bind Myc-Max recognition sitesCell, 1993
- Myc and Max proteins possess distinct transcriptional activitiesNature, 1992
- Loss of heterozygosity for 10q loci in human gliomasGenes, Chromosomes and Cancer, 1992
- Sequence-Specific DNA Binding by the c-Myc ProteinScience, 1990
- Involvement of the 'leucine zipper' region in the oligomerization and transforming activity of human c-myc proteinNature, 1989
- Mutation and Cancer: Statistical Study of RetinoblastomaProceedings of the National Academy of Sciences, 1971