Apoptosis of Retrogradely Degenerating Neurons Occurs in Association with the Accumulation of Perikaryal Mitochondria and Oxidative Damage to the Nucleus
Open Access
- 1 August 1998
- journal article
- Published by Elsevier in The American Journal of Pathology
- Vol. 153 (2) , 447-456
- https://doi.org/10.1016/s0002-9440(10)65588-5
Abstract
No abstract availableKeywords
This publication has 45 references indexed in Scilit:
- Prevention of Apoptosis by Bcl-2: Release of Cytochrome c from Mitochondria BlockedScience, 1997
- The Release of Cytochrome c from Mitochondria: A Primary Site for Bcl-2 Regulation of ApoptosisScience, 1997
- Why are mitochondria involved in apoptosis? Permeability transition pores and apoptosis as selective mechanisms to eliminate superoxide‐producing mitochondria and cellFEBS Letters, 1996
- Induction of Apoptotic Program in Cell-Free Extracts: Requirement for dATP and Cytochrome cPublished by Elsevier ,1996
- Mitochondrial control of nuclear apoptosis.The Journal of Experimental Medicine, 1996
- Oxidative DNA damage induced by simultaneous generation of nitric oxide and superoxideFEBS Letters, 1995
- Carbonyl‐Related Posttranslational Modification of Neurofilament Protein in the Neurofibrillary Pathology of Alzheimer's DiseaseJournal of Neurochemistry, 1995
- Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosisNature, 1993
- Transport of oxidative enzymes in nerve fibers; A histochemical investigation of the regenerative cycle in neuronsExperimental Neurology, 1959
- Electron microscopy of motor nerve cells following section of axonesThe Anatomical Record, 1954