A novel PPAR response element in the murine iNOS promoter
- 31 July 2005
- journal article
- Published by Elsevier in Molecular Immunology
- Vol. 42 (11) , 1303-1310
- https://doi.org/10.1016/j.molimm.2004.12.009
Abstract
No abstract availableKeywords
This publication has 26 references indexed in Scilit:
- Peroxisome Proliferation-Activated Receptor (PPAR)γ Is Not Necessary for Synthetic PPARγ Agonist Inhibition of Inducible Nitric-Oxide Synthase and Nitric OxideThe Journal of Pharmacology and Experimental Therapeutics, 2005
- Conditional Disruption of the Peroxisome Proliferator-Activated Receptor γ Gene in Mice Results in Lowered Expression of ABCA1, ABCG1, and apoE in Macrophages and Reduced Cholesterol EffluxMolecular and Cellular Biology, 2002
- Peroxisome proliferator–activated receptor γ agonists: Potential use for treating chronic inflammatory diseasesArthritis & Rheumatism, 2002
- 15-deoxy-Δ12,14-PGJ2 induces synoviocyte apoptosis and suppresses adjuvant-induced arthritis in ratsJournal of Clinical Investigation, 2000
- Peroxisome Proliferator-Activated Receptor γ-Dependent Repression of the Inducible Nitric Oxide Synthase GeneMolecular and Cellular Biology, 2000
- A novel therapy for colitis utilizing PPAR-γ ligands to inhibit the epithelial inflammatory responseJournal of Clinical Investigation, 1999
- Nuclear Integration of Glucocorticoid Receptor and Nuclear Factor-κB Signaling by CREB-binding Protein and Steroid Receptor Coactivator-1Published by Elsevier ,1998
- Potentiation of nitric oxide synthase expression by superoxide in interleukin 1β‐stimulated rat mesangial cellsFEBS Letters, 1998
- NITRIC OXIDE AND MACROPHAGE FUNCTIONAnnual Review of Immunology, 1997
- The role of nitric oxide in the pathogenesis of spontaneous murine autoimmune disease: increased nitric oxide production and nitric oxide synthase expression in MRL-lpr/lpr mice, and reduction of spontaneous glomerulonephritis and arthritis by orally administered NG-monomethyl-L-arginine.The Journal of Experimental Medicine, 1994