Dopamine (D2) or γ-aminobutyric acid (GABAB) receptor activation hyperpolarizes rat melanotrophs and pertussis toxin blocks these responses and the accompanying fall in [Ca2+]i

This publication has 16 references indexed in Scilit:

• Drug receptors and the inhibition of nerve cells
  British Journal of Pharmacology, 1989
• GABAA and GABAB receptors on porcine pars intermedia cells in primary culture: Functional role in modulating peptide release
  Neuroscience, 1986
• Novel synaptic responses mediated by dopamine and γ-aminobutyric acid in neuroendocrine cells of the intermediate pituitary
  Neuroscience Letters, 1986
• Pharmacological and ionic features of γ-aminobutyric acid receptors influencing electrical properties of melanotrophs isolated from the rat pars intermedia
  Neuroscience, 1985
• Effects of veratridine, tetrodotoxin and other drugs that alter electrical behaviour on secretion of melanocyte-stimulating hormone from melanotrophs of the pituitary pars intermedia
  Neuroscience, 1984
• GABA acts directly on cells of pituitary pars intermedia to alter hormone output
  Nature, 1983
• GABA Neuron Systems in Hypothalamus and the Pituitary Gland
  Neuroendocrinology, 1982
• Potassium-induced secretion of melanocytestimulating hormone from isolated pars intermedia cells signals participation of voltage-dependent calcium channels in stimulus-secretion coupling
  Neuroscience, 1981
• Lymphocyte membrane potential assessed with fluorescent probes
  Biochimica et Biophysica Acta (BBA) - Biomembranes, 1980
• The organization of tubero-hypophyseal and reticulo-infundibular catecholamine neuron systems in the rat brain
  Brain Research, 1973