Progression of Cerebral Amyloid Load Is Associated with the Apolipoprotein E ε4 Genotype in Alzheimer's Disease
- 2 July 2010
- journal article
- research article
- Published by Elsevier in Biological Psychiatry
- Vol. 68 (10) , 879-884
- https://doi.org/10.1016/j.biopsych.2010.05.013
Abstract
No abstract availableKeywords
Funding Information
- German Research Foundation (HE 4560/1-2, DR 445/3-1, DR 445/4-1)
- National Institutes of Health (R01 AG018402, R37 AG025516, P01 AG025204)
This publication has 40 references indexed in Scilit:
- Test-retest variability of quantitative [11C]PIB studies in Alzheimer’s diseaseEuropean Journal of Nuclear Medicine and Molecular Imaging, 2009
- Serial PIB and MRI in normal, mild cognitive impairment and Alzheimer's disease: implications for sequence of pathological events in Alzheimer's diseaseBrain, 2009
- Beta Amyloid in Alzheimer's Disease: Increased Deposition in Brain Is Reflected in Reduced Concentration in Cerebrospinal FluidBiological Psychiatry, 2009
- APOE ε4 allele predicts faster cognitive decline in mild Alzheimer diseaseNeurology, 2008
- Post-mortem correlates of in vivo PiB-PET amyloid imaging in a typical case of Alzheimer's diseaseBrain, 2008
- In vitro high affinity α‐synuclein binding sites for the amyloid imaging agent PIB are not matched by binding to Lewy bodies in postmortem human brain1Journal of Neurochemistry, 2008
- PIB is a non-specific imaging marker of amyloid-beta (A ) peptide-related cerebral amyloidosisBrain, 2007
- Imaging brain amyloid in Alzheimer's disease with Pittsburgh Compound‐BAnnals of Neurology, 2004
- Neuropathological stageing of Alzheimer-related changesActa Neuropathologica, 1991
- Clinical diagnosis of Alzheimer's diseaseNeurology, 1984