Hyperinsulinemia of the genetically obese (fa/fa) rat is decreased by a low dose of the 5-HT1A receptor agonist 8-hydroxy-2-(di-n-propylamino)tetralin (8-OH-DPAT)
- 1 February 1988
- journal article
- research article
- Published by Elsevier in European Journal of Pharmacology
- Vol. 147 (1) , 111-118
- https://doi.org/10.1016/0014-2999(88)90639-5
Abstract
No abstract availableThis publication has 34 references indexed in Scilit:
- Cardiovascular Response to 8-Hydroxy-2-(di-n-Propylamino) Tetralin (8-OH-DPAT) in the Rat: Site of Action and Pharmacological AnalysisJournal of Cardiovascular Pharmacology, 1987
- Motor Activity Increases Tryptophan, 5‐Hydroxyindoleacetic Acid, and Homovanillic Acid in Ventricular Cerebrospinal Fluid of the Conscious RatJournal of Neurochemistry, 1986
- 8-Hydroxy-2-(Di-n-propylamino)tetralin (8-OH-DPAT) elicits eating in free-feeding rats by acting on central serotonin neuronsEuropean Journal of Pharmacology, 1986
- Neurochemical mechanism of action of drugs which modify feeding via the serotoninergic systemAppetite, 1986
- Low doses of the putative serotonin agonist 8-hydroxy-2-(di-n-propylamino) tetralin (8-OH-DPAT) elicit feeding in the ratPsychopharmacology, 1985
- Serotonin and appetiteNeuropharmacology, 1984
- Probenecid sensitive pathway of elimination of dopamine and serotonin metabolites in CSF of the ratJournal Of Neural Transmission-Parkinsons Disease and Dementia Section, 1983
- Brain serotonergic activity and plasma amino acid levels in genetically obese Zucker ratsPharmacology Biochemistry and Behavior, 1982
- Catecholamines in discrete areas of the hypothalamus of obese and castrated male ratsPharmacology Biochemistry and Behavior, 1978
- Catecholamines in the brains of genetically obese ratsBrain Research, 1976