At the Innate Frontiers between Mother and Fetus: Linking Abortion with Complement Activation
- 28 February 2003
- Vol. 18 (2) , 169-172
- https://doi.org/10.1016/s1074-7613(03)00028-1
Abstract
No abstract availableKeywords
This publication has 18 references indexed in Scilit:
- CD1d and invariant NKT cells at the human maternal–fetal interfaceProceedings of the National Academy of Sciences, 2002
- Natural killer cells and pregnancyNature Reviews Immunology, 2002
- Inhibition of Allogeneic T Cell Proliferation by Indoleamine 2,3-Dioxygenase–expressing Dendritic CellsThe Journal of Experimental Medicine, 2002
- Removing the Vertebrate-Specific TBP N Terminus Disrupts Placental β2m-Dependent Interactions with the Maternal Immune SystemCell, 2002
- Complement C3 Activation Is Required for Antiphospholipid Antibody-induced Fetal LossThe Journal of Experimental Medicine, 2002
- MHC and MHC‐related proteins as pleiotropic signal moleculesThe FASEB Journal, 2001
- A Critical Role for Murine Complement Regulator Crry in Fetomaternal ToleranceScience, 2000
- Involvement of decidual Vα14 NKT cells in abortionProceedings of the National Academy of Sciences, 2000
- Much IDO about pregnancyNature Medicine, 1998
- Polymorphism of the human CD46 gene in normal individuals and in recurrent spontaneous abortionHuman Immunology, 1991