Effect of the Ionophore Monensin on Herpes Simplex Virus Type l-ïnduced Cell Fusion, Glycoprotein Synthesis, and Virion Infectivity

Abstract
The ionophore monensin inhibited the formation of mature, fully glycosylated glycoproteins gB, gC, and gD during herpes simplex virus type 1 infection of human embryonic lung cells. Underglycosylated forms, including the apparent high-mannose precursor forms of the major glycoproteins, appeared. Monensin inhibited virus-induced cell fusion. Infectious virions produced in the presence of monensin appeared to contain predominantly underglycosylated glycoproteins.

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