The Epstein–Barr Virus Latent Membrane Protein 1 Induces Interleukin-10 in Burkitt's Lymphoma Cells but Not in Hodgkin's Cells Involving the p38/SAPK2 Pathway
- 1 February 2001
- Vol. 280 (2) , 183-198
- https://doi.org/10.1006/viro.2000.0768
Abstract
No abstract availableKeywords
This publication has 58 references indexed in Scilit:
- The AT-rich region between −54 to −66 is important for the promoter activity of interleukin-10 in Epstein–Barr virus positive Burkitt’s lymphoma cellsGenes & Immunity, 1999
- Epstein–Barr virus latent membrane protein-1 (LMP1) signalling is distinct from CD40 and involves physical cooperation of its two C-terminus functional regionsOncogene, 1998
- THE ORIGIN OF HODGKIN AND REED/STERNBERG CELLS IN HODGKIN'S DISEASEAnnual Review of Immunology, 1998
- Epstein – Barr virus-encoded LMP1 and CD40 mediate IL-6 production in epithelial cells via an NF-κB pathway involving TNF receptor-associated factorsOncogene, 1997
- Normal and malignant B-cell development with special reference to Hodgkin's diseaseAnnals of Oncology, 1997
- The Epstein-Barr virus transforming protein LMP1 engages signaling proteins for the tumor necrosis factor receptor familyCell, 1995
- Prognostic value of serum IL‐10 and soluble IL‐2 receptor levels in aggressive non‐Hodgkin's lymphomaBritish Journal of Haematology, 1994
- Interleukin-10Annual Review of Immunology, 1993
- Epstein-Barr virus transformation induces B lymphocytes to produce human interleukin 10.The Journal of Experimental Medicine, 1993
- Interleukin 10(IL-10) inhibits cytokine synthesis by human monocytes: an autoregulatory role of IL-10 produced by monocytes.The Journal of Experimental Medicine, 1991