Mouse model made
- 1 February 1995
- journal article
- editorial
- Published by Springer Nature in Nature
- Vol. 373 (6514) , 476-477
- https://doi.org/10.1038/373476a0
Abstract
No abstract availableThis publication has 17 references indexed in Scilit:
- Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor proteinNature, 1995
- Making models for Alzheimer's diseaseNature Genetics, 1995
- Alzheimer's Disease: A Central Role for AmyloidJournal of Neuropathology and Experimental Neurology, 1994
- Apolipoprotein E Affects the Rate of Alzheimer Disease Expression: β-Amyloid Burden Is a Secondary Consequence Dependent on APOE Genotype and Duration of DiseaseJournal of Neuropathology and Experimental Neurology, 1994
- An Increased Percentage of Long Amyloid β Protein Secreted by Familial Amyloid β Protein Precursor (βApp 717 ) MutantsScience, 1994
- Introduction and expression of the 400 kilobase precursor amyloid protein gene in transgenic miceNature Genetics, 1993
- A68: a Major Subunit of Paired Helical Filaments and Derivatized Forms of Normal TauScience, 1991
- Segregation of a missense mutation in the amyloid precursor protein gene with familial Alzheimer's diseaseNature, 1991
- Amyloid plaque core protein in Alzheimer disease and Down syndrome.Proceedings of the National Academy of Sciences, 1985
- Alzheimer's disease: Initial report of the purification and characterization of a novel cerebrovascular amyloid proteinBiochemical and Biophysical Research Communications, 1984