Functional domains of APOBEC3G required for antiviral activity
- 5 May 2004
- journal article
- Published by Wiley in Journal of Cellular Biochemistry
- Vol. 92 (3) , 560-572
- https://doi.org/10.1002/jcb.20082
Abstract
No abstract availableKeywords
This publication has 17 references indexed in Scilit:
- The Human Immunodeficiency Virus Type 1 Vif Protein Reduces Intracellular Expression and Inhibits Packaging of APOBEC3G (CEM15), a Cellular Inhibitor of Virus InfectivityJournal of Virology, 2003
- HIV-1 Vif protein binds the editing enzyme APOBEC3G and induces its degradationNature Medicine, 2003
- Species-Specific Exclusion of APOBEC3G from HIV-1 Virions by VifCell, 2003
- DNA Deamination Mediates Innate Immunity to Retroviral InfectionCell, 2003
- Broad antiretroviral defence by human APOBEC3G through lethal editing of nascent reverse transcriptsNature, 2003
- Hypermutation of HIV-1 DNA in the Absence of the Vif ProteinScience, 2003
- Comprehensive Investigation of the Molecular Defect in vif -Deficient Human Immunodeficiency Virus Type 1 VirionsJournal of Virology, 2003
- An Anthropoid-Specific Locus of Orphan C to U RNA-Editing Enzymes on Chromosome 22Genomics, 2002
- Role of vif during Packing of the Core of HIV-1Virology, 1994
- The sor Gene of HIV-1 Is Required for Efficient Virus Transmission in VitroScience, 1987