Blockade of costimulatory molecules B7-1 (CD80) and B7-2 (CD86) down-regulates induction of contact sensitivity by haptenated epidermal cells
- 1 June 1997
- journal article
- Published by Oxford University Press (OUP) in British Journal of Dermatology
- Vol. 136 (6) , 846-852
- https://doi.org/10.1111/j.1365-2133.1997.tb03923.x
Abstract
No abstract availableThis publication has 25 references indexed in Scilit:
- Superantigen-induced cytokine expression in organ-cultured human skinJournal of Dermatological Science, 1996
- Low‐dose UVB radiation perturbs the functional expression of B7.1 and B7.2 co‐stimulatory molecules on human Langerhans cellsEuropean Journal of Immunology, 1995
- Immunological functions of non-professional antigen-presenting cells: new insights from studies of T-cell interactions with keratinocytesImmunology Today, 1994
- Expression of the B7/BB1 Activation Antigen and its Ligand CD28 in T-Cell-Mediated Skin DiseasesJournal of Investigative Dermatology, 1994
- Exaggerated and persistent cutaneous delayed-type hypersensitivity in transgenic mice whose epidermal keratinocytes constitutively express B7-1 antigen.Journal of Clinical Investigation, 1994
- The B7 and CD28 receptor familiesImmunology Today, 1994
- The Role of the CD28 Receptor During T Cell Responses to AntigenAnnual Review of Immunology, 1993
- Activated T cells induce expression of B7/BB1 on normal or leukemic B cells through a CD40-dependent signal.The Journal of Experimental Medicine, 1993
- Tumor Rejection After Direct Costimulation of CD8 + T Cells by B7-Transfected Melanoma CellsScience, 1993
- Functional expression of the costimulatory molecule, B7/BB1, on murine dendritic cell populations.The Journal of Experimental Medicine, 1992