The effect of TCR Vβ8 peptide protection and therapy on T cell populations isolated from the spinal cords of Lewis rats with experimental autoimmune encephalomyelitis

This publication has 22 references indexed in Scilit:

• Oral tolerance to myelin basic protein and natural recovery from experimental autoimmune encephalomyelitis are associated with downregulation of inflammatory cytokines and differential upregulation of transforming growth factor beta, interleukin 4, and prostaglandin E expression in the brain.
  The Journal of Experimental Medicine, 1992
• Direct demonstration of the infiltration of murine central nervous system by Pgp-1/CD44high CD45RBlow CD4+ T cells that induce experimental allergic encephalomyelitis
  Journal of Neuroimmunology, 1992
• T Cell Receptor Peptide Therapy Triggers Autoregulation of Experimental Encephalomyelitis
  Science, 1991
• Vaccination Against Experimental Allergic Encephalomyelitis with T Cell Receptor Peptides
  Science, 1989
• Peptide-specific prevention of experimental allergic encephalomyelitis. Neonatal tolerance induced to the dominant T cell determinant of myelin basic protein.
  The Journal of Experimental Medicine, 1989
• Clonal Expansion Versus Functional Clonal Inactivation: A Costimulatory Signalling Pathway Determines the Outcome of T Cell Antigen Receptor Occupancy
  Annual Review of Immunology, 1989
• The MRC OX-22- CD4+ T cells that help B cells in secondary immune responses derive from naive precursors with the MRC OX-22+ CD4+ phenotype.
  The Journal of Experimental Medicine, 1989
• Resistance to experimental autoimmune encephalomyelitis induced by neonatal tolerization to myelin basic protein: clonal elimination vs. regulation of autoaggressive lymphocytes
  European Journal of Immunology, 1989
• Both rat and mouse T cell receptors specific for the encephalitogenic determinant of myelin basic protein use similar V alpha and V beta chain genes even though the major histocompatibility complex and encephalitogenic determinants being recognized are different.
  The Journal of Experimental Medicine, 1989
• Loss of functional suppression is linked to decreases in circulating suppressor inducer (CD4 + 2H4 +) T Cells in multiple sclerosis
  Annals of Neurology, 1988