EVIDENCE FOR INVOLVEMENT OF HYPOCAPNIA AND HYPOPERFUSION IN AETIOLOGY OF NEUROLOGICAL DEFICIT AFTER CARDIOPULMONARY BYPASS
- 1 December 1987
- journal article
- research article
- Published by Elsevier in The Lancet
- Vol. 330 (8574) , 1493-1495
- https://doi.org/10.1016/s0140-6736(87)92624-9
Abstract
No abstract availableThis publication has 12 references indexed in Scilit:
- RETINAL MICROEMBOLISM DURING CARDIOPULMONARY BYPASS DEMONSTRATED BY FLUORESCEIN ANGIOGRAPHYThe Lancet, 1986
- BRAIN DAMAGE AFTER CARDIAC SURGERYThe Lancet, 1986
- Cerebral Carbon Dioxide Reactivity during Nonpulsatile Cardiopulmonary BypassThe Annals of Thoracic Surgery, 1986
- CEREBRAL CONSEQUENCES OF CARDIOPULMONARY BYPASSThe Lancet, 1986
- Early neurological complications of coronary artery bypass surgery.BMJ, 1985
- Brain hyperperfusion during cardiac operationsThe Journal of Thoracic and Cardiovascular Surgery, 1983
- Effects of decreasing arterial blood pressure on cerebral blood flow in the baboon. Influence of the sympathetic nervous system.Circulation Research, 1975
- Effect of carotid ligation on cerebral blood flow in baboons: 2. Response to hypoxia and haemorrhagic hypertensionJournal of Neurology, Neurosurgery & Psychiatry, 1974
- Pressure‐dependent cerebral ischemia during cardiopulmonary bypassNeurology, 1973
- Effect of alterations in the arterial carbon dioxide tension on the blood flow through the cerebral cortex at normal and low arterial blood pressures.Journal of Neurology, Neurosurgery & Psychiatry, 1965