Low-molecular-weight heparin prevents high glucose- and phorbol ester-induced TGF-β;1 gene activation
- 1 September 2001
- journal article
- Published by Elsevier in Kidney International
- Vol. 60 (3) , 935-943
- https://doi.org/10.1046/j.1523-1755.2001.060003935.x
Abstract
No abstract availableKeywords
This publication has 33 references indexed in Scilit:
- GLUT1 and TGF-β: the link between hyperglycaemia and diabetic nephropathyNephrology Dialysis Transplantation, 1999
- Evidence that TGF-β should be a therapeutic target in diabetic nephropathyKidney International, 1998
- Growth Factors and the Kidney in Diabetes MellitusCritical Reviews in Clinical Laboratory Sciences, 1998
- Characterization of protein kinase C beta isoform activation on the gene expression of transforming growth factor-beta, extracellular matrix components, and prostanoids in the glomeruli of diabetic rats.Journal of Clinical Investigation, 1997
- Treatment with a glycosaminoglycan formulation ameliorates experimental diabetic nephropathyKidney International, 1994
- Autocrine secretion of transforming growth factor-β in cultured rat mesangial cellsKidney International, 1992
- Glycosaminoglycans prevent morphological renal alterations and albuminuria in diabetic ratsKidney International, 1992
- Binding and internalization of heparin by vascular smooth muscle cellsJournal of Cellular Physiology, 1985
- Structural-functional relationships in diabetic nephropathy.Journal of Clinical Investigation, 1984
- A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye bindingAnalytical Biochemistry, 1976