Two α2-adrenergic receptor subtypes, α2A and α2C, inhibit transmitter release in the brain of gene-targeted mice

This publication has 29 references indexed in Scilit:

• Effect of G protein heterotrimer composition on coupling of neurotransmitter receptors to N-type Ca ²⁺ channel modulation in sympathetic neurons
  Proceedings of the National Academy of Sciences, 2000
• Two functionally distinct α2-adrenergic receptors regulate sympathetic neurotransmission
  Nature, 1999
• Abnormal Regulation of the Sympathetic Nervous System in α_2A-Adrenergic Receptor Knockout Mice
  Molecular Pharmacology, 1999
• Investigation of the subtypes of α2-adrenoceptor mediating prejunctional inhibition in rat atrium and cerebral cortex
  Naunyn-Schmiedebergs Archiv für experimentelle Pathologie und Pharmakologie, 1998
• Lack of autoreceptor-mediated inhibitory control of dopamine release in striatal synaptosomes of D2 receptor-deficient mice
  Brain Research, 1998
• Alterations in Dopamine Release But Not Dopamine Autoreceptor Function in Dopamine D₃Receptor Mutant Mice
  Journal of Neuroscience, 1998
• Subtype-Specific Intracellular Trafficking of α2-Adrenergic Receptors
  Molecular Pharmacology, 1997
• Central Hypotensive Effects of the α _2a -Adrenergic Receptor Subtype
  Science, 1996
• Cardiovascular Regulation in Mice Lacking α ₂ -Adrenergic Receptor Subtypes b and c
  Science, 1996
• Modulation of ion-channel function by G-protein-coupled receptors
  Trends in Neurosciences, 1994