Thalassaemia intermedia: a new molecular basis

Abstract
A 5-yr-old child heterozygous for .beta.-thalassemia has the clinical picture of thalassemia intermedia. Restriction endonuclease mapping shows that the child is homozygous for a triplicated .alpha. gene complex. The greater degree of globin chain imbalance resulting from 2 additional .alpha. chain genes is the likely mechanism for this unusually severe clinical phenotype.

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