Induction of Early Growth Response-1 Gene Expression by Calmodulin Antagonist Trifluoperazine through the Activation of Elk-1 in Human Fibrosarcoma HT1080 Cells
Open Access
- 1 March 2001
- journal article
- Published by Elsevier
- Vol. 276 (11) , 7797-7805
- https://doi.org/10.1074/jbc.m009465200
Abstract
No abstract availableKeywords
This publication has 36 references indexed in Scilit:
- The Transcription Factor EGR-1 Suppresses Transformation of Human Fibrosarcoma HT1080 Cells by Coordinated Induction of Transforming Growth Factor-β1, Fibronectin, and Plasminogen Activator Inhibitor-1Journal of Biological Chemistry, 1999
- Decreased Egr-1 expression in human, mouse and rat mammary cells and tissues correlates with tumor formationInternational Journal of Cancer, 1997
- Integration of MAP Kinase Signal Transduction Pathways at the Serum Response ElementScience, 1995
- SIGNAL TRANSMISSION BETWEEN THE PLASMA MEMBRANE AND NUCLEUS OF T LYMPHOCYTESAnnual Review of Biochemistry, 1994
- Activated N-ras controls the transformed phenotype of HT1080 human fibrosarcoma cellsCell, 1987
- Calcium ions and the control of proliferation in normal and cancer cellsBioscience Reports, 1982
- Increased content of calmodulin in morris hepatoma 5123 t.c.(h)Biochemical and Biophysical Research Communications, 1981
- Damage of cellular functions by trifluoperazine, a calmodulin-specific drugExperimental Cell Research, 1980
- Calmodulin—an intracellular calcium receptorNature, 1980
- Calmodulin Plays a Pivotal Role in Cellular RegulationScience, 1980