Amphetamine-Induced Fos is Reduced in Limbic Cortical Regions but not in the Caudate or Accumbens in a Genetic Model of NMDA Receptor Hypofunction
Open Access
- 29 September 2004
- journal article
- research article
- Published by Springer Nature in Neuropsychopharmacology
- Vol. 29 (12) , 2180-2188
- https://doi.org/10.1038/sj.npp.1300548
Abstract
No abstract availableKeywords
This publication has 52 references indexed in Scilit:
- Deficits in sensorimotor gating and tests of social behavior in a genetic model of reduced NMDA receptor functionBehavioural Brain Research, 2004
- Subchronic Continuous Phencyclidine Administration Potentiates Amphetamine-Induced Frontal Cortex Dopamine ReleaseNeuropsychopharmacology, 2003
- Alterations in regional brain metabolism in genetic and pharmacological models of reduced NMDA receptor functionBrain Research, 2002
- Reversal of Phencyclidine Effects by a Group II Metabotropic Glutamate Receptor Agonist in RatsScience, 1998
- Ketamine-Induced Exacerbation of Psychotic Symptoms and Cognitive Impairment in Neuroleptic-Free SchizophrenicsNeuropsychopharmacology, 1997
- Targeted disruption of NMDA receptor 1 gene abolishes NMDA response and results in neonatal deathPublished by Elsevier ,1994
- Whisker-related neuronal patterns fail to develop in the trigeminal brainstem nuclei of NMDAR1 knockout micePublished by Elsevier ,1994
- Molecular Diversity of Glutamate Receptors and Implications for Brain FunctionScience, 1992
- MK801 induces c-fos protein in thalamic and neocortical neurons of rat brainNeuroscience Letters, 1990
- The Effect of 1-Arylcyclohexylamine (Sernyl) on Twelve Normal VolunteersJournal of Mental Science, 1960