Temporal activation of c‐Jun N‐terminal kinase in adult transgenic heart via cre‐loxP‐mediated DNA recombination
Open Access
- 19 February 2003
- journal article
- fj express-summaries
- Published by Wiley in The FASEB Journal
- Vol. 17 (6) , 749-751
- https://doi.org/10.1096/fj.02-0438fje
Abstract
Using a cre-loxP-mediated gene-switch approach, we achieved targeted JNK activation in adult hearts. A transgenic model is established carrying a floxed gene-switch construct that directs GFP marker ...Keywords
Funding Information
- American Heart Association
- National Institutes of Health
This publication has 21 references indexed in Scilit:
- Regulation of cardiac hypertrophy in vivo by the stress-activated protein kinases/c-Jun NH2-terminal kinasesJournal of Clinical Investigation, 1999
- Comparison of the Tamoxifen Regulated Chimeric Cre Recombinases MerCreMer and CreMerBiological Chemistry, 1999
- “Stress-Responsive” Mitogen-Activated Protein Kinases (c-Jun N-Terminal Kinases and p38 Mitogen-Activated Protein Kinases) in the MyocardiumCirculation Research, 1998
- Timing Is Everything in LifeCirculation Research, 1998
- Stimulation of “Stress-regulated” Mitogen-activated Protein Kinases (Stress-activated Protein Kinases/c-Jun N-terminal Kinases and p38-Mitogen-activated Protein Kinases) in Perfused Rat Hearts by Oxidative and Other StressesJournal of Biological Chemistry, 1998
- Conditional Transgene Expression in the HeartCirculation Research, 1996
- Conditional gene targeting.Journal of Clinical Investigation, 1996
- Stress-Activated Protein Kinases in Cardiovascular DiseaseCirculation Research, 1996
- Transgenic Remodeling of the Contractile Apparatus in the Mammalian HeartCirculation Research, 1996
- Cellular Stresses Differentially Activate c-Jun N-terminal Protein Kinases and Extracellular Signal-regulated Protein Kinases in Cultured Ventricular MyocytesJournal of Biological Chemistry, 1995