Regulation of Staphylococcus epidermidis-induced IFN-γ in whole human blood: the role of endogenous IL-18, IL-12, IL-1, and TNF
- 21 January 2003
- Vol. 21 (2) , 65-73
- https://doi.org/10.1016/s1043-4666(02)00501-x
Abstract
No abstract availableKeywords
This publication has 43 references indexed in Scilit:
- Human Host Defense and Cytokines in Mycobacterial Infectious Diseases: Interleukin‐18 Cannot Compensate for Genetic Defects in the Interleukin‐12 SystemClinical Infectious Diseases, 2002
- Tuberculosis Associated with Infliximab, a Tumor Necrosis Factor α–Neutralizing AgentNew England Journal of Medicine, 2001
- Differential Roles of Interleukin-18 (IL-18) and IL-12 for Induction of Gamma Interferon by Staphylococcal Cell Wall Components and SuperantigensInfection and Immunity, 2001
- Invasive Pulmonary Aspergillosis Associated with Infliximab TherapyNew England Journal of Medicine, 2001
- Immunomodulatory Role of Endogenous Interleukin-18 in Gamma Interferon-Mediated Resolution of ReplicativeLegionella pneumophilaLung InfectionInfection and Immunity, 2000
- Interleukin‐18 Enhances Lipopolysaccharide‐Induced Interferon‐γ Production in Human Whole Blood CulturesThe Journal of Infectious Diseases, 1998
- Severe Mycobacterial and Salmonella Infections in Interleukin-12 Receptor-Deficient PatientsScience, 1998
- TUMOR NECROSIS FACTOR IN SEPSIS: MEDIATOR OF MULTIPLE ORGAN FAILURE OR ESSENTIAL PART OF HOST DEFENSE?Shock, 1995
- Interleukin 1 induces a shock-like state in rabbits. Synergism with tumor necrosis factor and the effect of cyclooxygenase inhibition.Journal of Clinical Investigation, 1988
- Toxic-Shock SyndromeNew England Journal of Medicine, 1980