EFFECT OF HOMOCYSTEINE ON THE PRODUCTION OF NITRIC OXIDE IN ENDOTHELIAL CELLS
- 7 October 1999
- journal article
- research article
- Published by Wiley in Clinical and Experimental Pharmacology and Physiology
- Vol. 26 (10) , 817-818
- https://doi.org/10.1046/j.1440-1681.1999.03133.x
Abstract
1. Homocysteine decreased the release of nitric oxide in cultured human umbilical vein endothelial cells. 2. Homocysteine did not affect constitutive and inducible nitric oxide synthase activity.Keywords
This publication has 19 references indexed in Scilit:
- The oxidant stress of hyperhomocyst(e)inemia.Journal of Clinical Investigation, 1996
- Vascular dysfunction in monkeys with diet-induced hyperhomocyst(e)inemia.Journal of Clinical Investigation, 1996
- A quantitative assessment of plasma homocysteine as a risk factor for vascular disease. Probable benefits of increasing folic acid intakesPublished by American Medical Association (AMA) ,1995
- The Emerging Multifaceted Roles of Nitric OxideAnnals of Surgery, 1995
- Homocyst(e)ine and arterial occlusive diseasesJournal of Internal Medicine, 1994
- Endothelial nitric oxide and cardiovascular diseaseJournal of Internal Medicine, 1994
- Induction of nitric oxide synthase gene by interleukin in vascular smooth muscle cells.Hypertension, 1993
- Adverse vascular effects of homocysteine are modulated by endothelium-derived relaxing factor and related oxides of nitrogen.Journal of Clinical Investigation, 1993
- Plasma homocysteine before and after methionine loading with regard to age, gender, and menopausal statusEuropean Journal of Clinical Investigation, 1992
- Hyperhomocysteinemia: An Independent Risk Factor for Vascular DiseaseNew England Journal of Medicine, 1991