OBSERVATIONS ON THE LATE EFFECTS OF DENERVATION OF THE CAROTID SINUSES AND SECTION OF THE DEPRESSOR NERVES

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Abstract
In 3 dogs with the cardio-aortic depressor mechanisms destroyed for 1-11 mos., the respiratory responses to inhalation of increasing concs. of COa were less marked than those of normal dogs. In acute terminal expts., no evidence of regeneration of the carotid sinus or depressor nerves was obtained. The blood pressure effects of inhalation of amyl nitrite and asphyxia were qualitatively normal.

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